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 veryhotthread  Author  Topic: Stuff & Nonsense  (Read 10733 times)
WingsofCrystal
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« Reply #12840 on: Jun 4th, 2015, 09:10am »

GOOD MORNING Z & ALL OF OUR UFOCASEBOOKERS cheesy

~

Science Daily

Researchers find speedometer in the brain

Date: June 3, 2015
Source: DZNE - German Center for Neurodegenerative Diseases

Scientists at the German Center for Neurodegenerative Diseases (DZNE) and the University of Bonn led by Prof. Stefan Remy report on this in the journal Neuron. Their investigations give new insights into the workings of spatial memory. Furthermore, they could also help improve our understanding of movement related symptoms associated with Parkinson's disease.

In a familiar environment our movements are purposeful. For example, if we leave our office desk for a coffee break, we naturally follow a predefined route that has been stored in our memory: Through the office door, left into the hall, past the windows. To keep us on track, our brain has to process varying sensory impressions quickly. "This is a fundamental issue our brain has to deal with. Not just on our way to the coffee machine, but any time we move in space. For example when we are on a bike or in a car," explains Remy. With increasing speed, the data rate also increases, he emphasizes: "The faster we move, the less time the brain has to take in environmental cues and to associate them with a location on our memorized spatial map. Our perception therefore has to keep pace with the speed of movement so that we remember the right way to go. Otherwise we end up at the copy machine instead of the coffee machine."

Rhythmic fluctuations

It has been known for some time that the hippocampus -- the part of the brain that controls memory, particularly spatial memory -- adjusts to the speed of locomotion. "The electrical activity of the hippocampus undergoes rhythmic fluctuations. The faster we move, the faster certain nerve cells are activated," says Remy. "This increased activation rate sensitizes the brain. It becomes more receptive to the changing sensory impressions that have to be processed when moving."

But how does the brain actually know how fast a movement is? Previously there was no answer to this question. Now, Remy and his colleagues have decoded the mechanism. For this, they stimulated specific areas within the mouse brain and recorded the ensuing brain activity and the mice's locomotion. "We have identified the neural circuits in mice that link their spatial memory to the speed of their movement. This interplay is an important foundation for a functioning spatial memory," says Remy. "We assume that humans have similar nerve cells, as the brains of mice and humans have a very similar structure in these regions."

Small cell group

The cells in question are located in the "medial septum," a part of the brain directly connected to the hippocampus. They make up a relatively small group comprising a few thousand cells. "They gather information from sensory and locomotor systems, determine the speed of movement and transmit this information to the hippocampus. In this way, they tune the spatial memory systems for optimized processing of sensory stimuli during locomotion," explains Remy. However, these circuits have even more functions. "We have found that they also give the start signal for locomotion and that they actively control its speed. Until now, this control function was almost exclusively ascribed to the motor cerebral cortex."

These newly discovered nerve cells are linked with areas of the brain that are affected by Parkinson's in humans. This disease is associated with movement-related symptoms and can cause dementia. "In this respect, our results go beyond the workings of spatial memory; they also have the potential to provide new insights into how memory systems and the execution of movements are affected in Parkinson's disease," says Remy.

http://www.sciencedaily.com/releases/2015/06/150603132252.htm#

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« Reply #12841 on: Jun 4th, 2015, 4:44pm »

TRUE HEROES:


https://www.youtube.com/embed/7tyT4glkvBs


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« Reply #12842 on: Jun 5th, 2015, 09:24am »

Thank you Swamprat.

Good morning UFOCasebookers.

Please send up prayers for Nichelle Nichols.

~

82-year-old ‘Star Trek’ actress Nichelle Nichols suffers stroke


By HENRY HANKS
CNN
June 4, 2015 - 9:26pm


Barely three months after the loss of Leonard Nimoy, “Star Trek” fans have gotten more troubling news.

Nichelle Nichols, 82, who played Lt. Nyota Uhura in the original TV series and films, suffered a mild stroke Wednesday night, according to a post Thursday on her Facebook page.

“Last night while at her home in L.A., Nichelle Nichols suffered from a mild stroke,” said the statement by Zach McGinnis of Galactic Productions, which books Nichols and other actors for public appearances.

“She is currently undergoing testing to determine how severe the stroke was,” the statement continued. Please keep her in your thoughts.”

Nichols was a TV rarity when “Star Trek” began in 1966: a black woman in a notable role on a prime-time series. There had been African-American women on TV before, but they often played maids or domestics; Nichols’ Uhura was an integral part of the multicultural “Star Trek” crew.

The Rev. Martin Luther King Jr. called it “the first nonstereotypical role portrayed by a black woman in television history.”

Actor George Takei, Nichols’ “Star Trek” compatriot, sent along his best wishes via Twitter on Thursday. “We don’t have any diagnosis on her condition but our thoughts are with her,” he said.

McGinnis updated CNN Thursday evening on her condition.

“CAT scan came back negative, and we are awaiting the results from the MRI,” he said.

“Currently she is awake, eating, in good spirits and able to have full conversations. Her right side has shown minor signs or mobility loss, but she is not showing any signs of paralysis.”

http://www.reviewjournal.com/entertainment/the-reel/82-year-old-star-trek-actress-nichelle-nichols-suffers-stroke

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« Reply #12843 on: Jun 5th, 2015, 10:48am »







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« Reply #12844 on: Jun 6th, 2015, 06:52am »

GOOD SATURDAY MORNING ALL cheesy

This is worth looking at:

Popular Mechanics

Jun 5, 2015 @ 3:24 PM

The Most Hilarious Robo-Falls from the DARPA Robotics Challenge

More like DERPA robotics challenge

By Eric Limer

gallery after the jump:
http://www.popularmechanics.com/technology/robots/a15907/best-falls-from-darpa-robot-challenge/

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GREAT SPIRITS ALWAYS ENCOUNTER THE MOST VIOLENT OPPOSITION FROM MEDIOCRE MINDS E=MC2


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« Reply #12845 on: Jun 6th, 2015, 07:19am »

GOOD MORNING VIETNAM ~ CRYSTAL ~ CASEBOOK ~ cool

HA LONG BAY, VIETNAM...

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« Reply #12846 on: Jun 6th, 2015, 07:27am »

GOOD MORNING Z grin

CRYSTAL

~





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« Reply #12847 on: Jun 6th, 2015, 9:13pm »

AHHHHHHH SOOOOOOO ~ HAL SON...

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« Reply #12848 on: Jun 7th, 2015, 07:45am »

WELL...CAUGHT YA PEEKIN ~ cool

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EDIT TO ADD:

grin

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« Reply #12849 on: Jun 7th, 2015, 08:45am »

GOOD MORNING grin






CRYSTAL



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« Reply #12850 on: Jun 7th, 2015, 5:48pm »

Facebook use linked to this mental illness

Have you been feeling a little blue lately? Well, if you use Facebook heavily, that may be why. Recently, an extensive study of users in Poland found that excessive Facebook surfing is linked to mental illness such as depression, sexual dysfunction and a variety of other, more serious, addictions. The researchers even coined a term for such use, calling it "Facebook intrusion."

Facebook intrusion occurs when you spend enough time on the site that it interferes with your daily activities, personal relationships and routines. This type of addiction is now well-documented and can possibly be the reason why there has been a jump in depression diagnoses over the last 10 years. So next time you find yourself staring blankly at your Facebook screen, put it down and take a walk. It might save your sanity!

The study was conducted with over 600 participants, the average age being 28. The researchers found people that used Facebook heavily are more likely to be diagnosed with clinical depression than people who use the site moderately. They also found that young males are more likely to suffer from Internet-based depression than any other group.

Online depression partially stems from the constant need to compare oneself to the events, pictures and news being uploaded daily from Facebook "friends." Introverts are also drawn to this kind of depression due to their overall lack of social skills. They turn to the Internet as a way to create a fictitious online persona, often making up details about their life that are nowhere close to the truth. The false sense of personal interaction is also a factor that plays into Facebook depression.

Some symptoms of Facebook depression are a constant urge to fiddle with your profile, crippling anxiety if you don't update your status at least four times a day and if you have created a separate profile for your furry friend. Having a large number of Facebook friends also indicates a Facebook addiction. If you've never met at least half of your friend group then you need to consider the possibility that you are addicted to Facebook.

If this sounds like you, check the amount of time you spend surfing around on Facebook. If it seems excessive then take steps to actively cut down on the amount of time you spend on the site. Depression can be warded off with doing easy things like walking for 30 minutes, getting more sleep, playing with animals and forcing yourself to smile more.

http://www.komando.com/happening-now/310986/facebook-use-linked-to-this-mental-illness/2

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« Reply #12851 on: Jun 8th, 2015, 07:00am »

GOOD MORNING CRYSTAL ~ CASEBOOK cool

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« Reply #12852 on: Jun 8th, 2015, 07:50am »

Hey Swamprat,


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Stay on Facebook too long and you will end up like Kenny grin

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« Reply #12853 on: Jun 8th, 2015, 07:55am »

on Jun 8th, 2015, 07:00am, ZETAR wrote:
GOOD MORNING CRYSTAL ~ CASEBOOK cool

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GOOD MORNING Z & ALL OF OUR UFOCASEBOOKERS cheesy


~

Wired

Author: Kim Zetter
Date of Publication: 06.08.15.

Hacker Can Send Fatal Dose to Hospital Drug Pumps

When security researcher Billy Rios reported earlier this year that he’d found vulnerabilities in a popular drug infusion pump that would allow a hacker to raise the dosage limit on medication delivered to patients, there was little cause for concern.

Altering the allowable limits of a particular drug simply meant that if a caregiver accidentally instructed the pump to give too high or too low a dosage, the pump wouldn’t issue an alert. This seemed much less alarming than if the pumps had vulnerabilities that would allow a hacker to actually alter the dosage itself.

Now Rios says he’s found the more serious vulnerabilities in several models of pumps made by the same manufacturer, which would allow a hacker to surreptitiously and remotely change the amount of drugs administered to a patient.

“This is the first time we know we can change the dosage,” Rios told WIRED.

The vulnerabilities are known to affect at least five models of drug infusion pumps made by Hospira—an Illinois firm with more than 400,000 intravenous drug pumps installed in hospitals around the world.

The vulnerable models include the company’s standard PCA LifeCare pumps; its PCA3 LifeCare and PCA5 LifeCare pumps; its Symbiq line of pumps, which Hospira stopped selling in 2013 due to concerns raised by the FDA over other quality and safety issues with the pumps; and its Plum A+ model of pumps. Hospira has at least 325,000 of the latter model alone installed in hospitals worldwide.

These are the systems that Rios knows are vulnerable because he’s tested them. But he suspects that the company’s Plum A+3 and its Sapphire and SapphirePlus models are equally vulnerable too.

Hospira did not respond to a request for comment.

Earlier this year, Rios went public with information about a different security issue with Hospira’s LifeCare pumps.

This one involved drug libraries used with the pumps, which help set upper and lower boundaries for dosages of intravenous drugs a pump can safely administer. Because the libraries don’t require authentication, Rios found that anyone on the hospital’s network—including patients in the hospital or a hacker accessing the pumps over the Internet—can load a new drug library that alters the limits for a drug.

At the time he publicly disclosed the library vulnerability, Rios told WIRED that he had not yet found any vulnerabilities that would allow him to actually alter a drug dosage, though he was working on it. But he now acknowledges that he had found these more serious vulnerabilities in the LifeCare pumps at the time and had in fact reported them to Hospira and the FDA last year. At the time he hadn’t yet tested a Plum A+ pump, however.

The new vulnerabilities would allow attackers to remotely alter the firmware on the pumps, giving them complete control of the devices and the ability to alter dosages delivered to patients. And because the pumps are also vulnerable to the previous library vulnerability he disclosed, an attacker would be able to first raise the dosage above the maximum limit before delivering a potentially deadly dosage without the pump issuing an alert.

How the Firmware Security Flaw Works

The problem lies with a communication module in the LifeCare and Plum A+ pumps. Hospitals use the communication modules to update the libraries on the pumps. But the communication modules are connected via a serial cable to a circuit board in the pumps, which contains the firmware. Hospira uses this serial connection to remotely access the firmware and update it. But hackers can use it for the same purpose.

The serial connection would be less of a concern if Hospira’s pumps accepted only legitimate firmware updates that were authenticated and digitally signed. But Rios says they’ll accept any update, which means anyone can alter the software on the pumps.

“And if you can update the firmware on the main board, you can make the pump do whatever you like,” Rios says.

A hacker could not only change the dosage of drugs delivered to a patient but also alter the pump’s display screen to indicate a safe dosage was being delivered.

more after the jump:
http://www.wired.com/2015/06/hackers-can-send-fatal-doses-hospital-drug-pumps/

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« Reply #12854 on: Jun 8th, 2015, 07:57am »






Published on Jun 8, 2015

~

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